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|Tested species reactivity||Human, Primate|
|Published species reactivity||Human|
|Host / Isotype||Mouse / IgM|
|Immunogen||T cell line HPB-ALL.|
|Storage buffer||TBS with 0.2% BSA|
|Contains||15mM sodium azide|
|Storage Conditions||4° C, store in dark, DO NOT FREEZE!|
|Tested Applications||Dilution *|
|Flow Cytometry (Flow)||Assay Dependent|
* Suggested working dilutions are given as a guide only. It is recommended that the user titrate the product for use in their own experiment using appropriate negative and positive controls.
|Immunohistochemistry (IHC)||See 1 publications below|
CD55 (decay-accelerating factor, DAF) is a GPI-anchored membrane glycoprotein that protects autologous cells from classical and alternative pathway of complement cascade. Bidirectional interactions between CD55 and CD97 are involved in T cell regulation and CD55 can still regulate complement when bound to CD97. In tumours, besides protection agains complement, CD55 promotes neoangiogenesis, tumorigenesis, invasiveness and evasion of apoptosis.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
Disease-specific and inflammation-independent stromal alterations in spondylarthritis synovitis.
MA1-19573 was used in immunohistochemistry to identify novel disease-specific pathways in spondylarthritis synovitis using geneome-wide microarray analysis
|Yeremenko N,Noordenbos T,Cantaert T,van Tok M,van de Sande M,Cañete JD,Tak PP,Baeten D||Arthritis and rheumatism (65:174)||2013|
CD55 antigen; CD55 molecule, decay accelerating factor for complement (Cromer blood group); CROM; DAF; RP11-357P18.1; TC
CD55; CR; CROM; DAF; TC