Note: You clicked on an external link, which has been disabled in order to keep your shopping session open.
|Tested species reactivity||Virus|
|Published species reactivity||Virus, Not Applicable|
|Host / Isotype||Mouse / IgG1|
|Immunogen||Influenza A / Puerto Rico / 8 / 34 (H1N1) and A/Bangkok / 1 / 79 (H3N2) viruses.|
|Storage buffer||PBS, pH 7.5|
|Contains||0.09% sodium azide|
|Storage Conditions||4°C or -20°C if preferred|
|Tested Applications||Dilution *|
|Immunohistochemistry (Paraffin) (IHC (P))||Assay Dependent|
|Western Blot (WB)||Assay Dependent|
* Suggested working dilutions are given as a guide only. It is recommended that the user titrate the product for use in their own experiment using appropriate negative and positive controls.
|Western Blot (WB)||See 2 publications below|
Purity is >90% IgG content by SDS PAGE.
The M1 protein of influenza A virus has multiple regulatory functions during the infectious cycle, which include mediation of nuclear export of viral ribonucleoproteins, inhibition of viral transcription and a crucial role in virus assembly and budding. The filamentous phenotype was lost when the amino acid at position 41 was switched from A to V. This observation suggests that an interaction among these regions of M1 may occur during assembly.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
Identification of Influenza A Virus PB2 Residues Involved in Enhanced Polymerase Activity and Virus Growth in Mammalian Cells at Low Temperatures.
MA1-80736 was used in western blot to study avian viral polymerase mutants.
|Hayashi T,Wills S,Bussey KA,Takimoto T||Journal of virology (89:8042)||2015|
|Not Applicable||Not Cited||
Recruitment of RED-SMU1 complex by Influenza A Virus RNA polymerase to control Viral mRNA splicing.
MA1-80736 was used in western blot to determine viral mRNA splicing controlled by the recruitment of RED-SMU1 complex by Influenza A Virus RNA polymerase
|Fournier G,Chiang C,Munier S,Tomoiu A,Demeret C,Vidalain PO,Jacob Y,Naffakh N||PLoS pathogens (10:null)||2014|