|Tested species reactivity||Human|
|Published species reactivity||Human, Mouse|
|Host / Isotype||Mouse / IgG1|
|Immunogen||CATQRLANFLV coupled to tuberculin|
|Storage buffer||tissue culture supernatant|
|Contains||0.09% sodium azide|
|Storage Conditions||4°C or -20°C if preferred|
|Tested Applications||Dilution *|
|Immunohistochemistry (Frozen) (IHC (F))||Assay dependent|
|Immunohistochemistry (Paraffin) (IHC (P))||Assay dependent|
* Suggested working dilutions are given as a guide only. It is recommended that the user titrate the product for use in their own experiment using appropriate negative and positive controls.
|Immunohistochemistry (IHC)||See 3 publications below|
MA1-34685 detects Islet amylin peptide from human samples.
MA1-34685 has been successfully used in immunohistochemistry (frozen tissue), and immunohistochemistry (paraffin tissue) applications.
The MA1-34685 immunogen is CATQRLANFLV coupled to tuberculin.
Store at 4°C or -20°C if preferred. Avoid Freeze/Thaw Cycles.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
Diabetes mellitus in pancreatic cancer: is it a causal relationship?
MA1-34685 was used in immunohistochemistry to examine the relationship between diabetes mellitus and pancreatic cancer
|Katsumichi I,Pour PM||American journal of surgery (194:S71)||2007|
Distribution of pancreatic endocrine cells including IAPP-expressing cells in non-diabetic and type 2 diabetic cases.
MA1-34685 was used in immunohistochemistry to investigate the distribution of pancreatic endocrine cells in non-diabetic and diabetic individuals
|Iki K,Pour PM||The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society (55:111)||2007|
Downregulation of EGF receptor signaling in pancreatic islets causes diabetes due to impaired postnatal beta-cell growth.
MA1-34685 was used in immunohistochemistry to study the role of EGF receptor signaling in postnatal beta-cell growth in pancreatic islets
|Miettinen PJ,Ustinov J,Ormio P,Gao R,Palgi J,Hakonen E,Juntti-Berggren L,Berggren PO,Otonkoski T||Diabetes (55:3299)||2006|