Immunofluorescent analysis of Sodium/Calcium Exchanger using Anti-Sodium/Calcium Exchanger Monoclonal Antibody (C2C12) (Product# MA3-926) shows staining in A2058 Cells. Sodium/Calcium Exchanger staining (green), F-Actin staining with Phalloidin (red) and nuclei with DAPI (blue) is shown. Cells were grown on chamber slides and fixed with formaldehyde prior to staining. Cells were probed without (control) or with or an antibody recognizing Sodium/Calcium Exchanger (Product# MA3-926) at a dilution of 1:100 over night at 4 °C, washed with PBS and incubated with a DyLight-488 conjugated secondary antibody (Product# 35503, Goat Anti-Mouse). Images were taken at 60X magnification.
|Tested species reactivity||Dog, Guinea pig, Human, Mouse, Rabbit, Rat|
|Published species reactivity||Dog, Rabbit, Rat, Pig, Rodent, Marsupial, Bovine, Mouse, Human, Not Applicable, Guinea pig|
|Host / Isotype||Mouse / IgM|
|Immunogen||Purified canine cardiac sodium/calcium exchanger.|
|Contains||0.05% sodium azide|
|Storage Conditions||-20° C, Avoid Freeze/Thaw Cycles|
|Tested Applications||Dilution *|
|Flow Cytometry (Flow)||1:20|
|Immunohistochemistry (Frozen) (IHC (F))||Assay Dependent|
|Immunohistochemistry (Paraffin) (IHC (P))||1/100|
|Immunoprecipitation (IP)||Assay dependent|
|Western Blot (WB)||1:1,000|
* Suggested working dilutions are given as a guide only. It is recommended that the user titrate the product for use in their own experiment using appropriate negative and positive controls.
MA3-926 detects sodium/calcium exchanger from human, canine, rabbit, mouse, guinea pig and rat kidney and cardiac tissues.
MA3-926 has been successfully used in Western blot, immunofluorescence, immunohistochemistry, and immunoprecipitation procedures. By Western blot, this antibody detects a 120 kDa protein representing the sodium/calcium exchanger from guinea pig cardiac extract. The bands seen at 70 kDa and 160 kDa represent a proteolytic fragment and non-reduced exchanger respectively. MA3-926 is not recommended for Western blot procedures of rat tissues. Immunohistochemical staining of the sodium/calcium exchanger in rat heart with MA3-926 results in staining of the plasma membrane and intense staining of cardiac T-tubular membrane.
The MA3-926 antigen is purified canine cardiac sodium/calcium exchanger. This antibody recognizes an epitope between amino acids 371-525, which is on the intracellular side of the plasma membrane.
The sodium/calcium exchanger of cardiac sarcolemma rapidly transports calcium during excitation-contraction coupling and is the dominant myocardial calcium efflux mechanism. The sodium/calcium exchanger uses the transmembrane sodium gradient to catalyze countertransport of calcium against its electrochemical gradient in a 3 sodium : 1 calcium exchange. Sodium/calcium exchange activity is present in excitable cells and in non-excitable cells.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
Calcium/calmodulin-dependent kinase II and nitric oxide synthase 1-dependent modulation of ryanodine receptors during ß-adrenergic stimulation is restricted to the dyadic cleft.
MA3-926 was used in immunocytochemistry to characterize modulation of ryanodine receptors during beta-adrenergic stimulation and restriction to the dyadic cleft via nictric oxide synthase-1 and calcium/calmodulin-dependent kinase II
|Dries E,Santiago DJ,Johnson DM,Gilbert G,Holemans P,Korte SM,Roderick HL,Sipido KR||The Journal of physiology (594:5923)||2016|
Dystrophic cardiomyopathy: role of TRPV2 channels in stretch-induced cell damage.
MA3-926 was used in immunocytochemistry to study if transient receptor potential vanilloid channels type 2 form subunits of the dysregulated stretch-activated channels in cardiac dystrophy
|Lorin C,Vögeli I,Niggli E||Cardiovascular research (106:153)||2015|
Hierarchical clustering of ryanodine receptors enables emergence of a calcium clock in sinoatrial node cells.
MA3-926 was used in immunocytochemistry to study and model the role of ryanodine receptors in the sinoatrial node calcium clock mechanism
|Stern MD,Maltseva LA,Juhaszova M,Sollott SJ,Lakatta EG,Maltsev VA||The Journal of general physiology (143:577)||2014|
Preservation of cardiomyocytes from the adult heart.
MA3-926 was used in immunocytochemistry to investigate inhibition of myosin II ATPase as a mechanism for preserving isolated adult cardiomyocytes
|Abi-Gerges N,Pointon A,Pullen GF,Morton MJ,Oldman KL,Armstrong D,Valentin JP,Pollard CE||Journal of molecular and cellular cardiology (64:108)||2013|
Distribution patterns of the Na+-Ca2+ exchanger and caveolin-3 in developing rabbit cardiomyocytes.
MA3-926 was used in immunocytochemistry to investigate the level of sodium/calcium exchanger and caveolin-3 in heart cells during development
|Lin E,Hung VH,Kashihara H,Dan P,Tibbits GF||Cell calcium (45:369)||2009|
Three-dimensional distribution of cardiac Na+-Ca2+ exchanger and ryanodine receptor during development.
MA3-926 was used in immunocytochemistry to study the localization and functions of the cardiac Na+-Ca2+ exchanger and ryanodine receptor during development
|Dan P,Lin E,Huang J,Biln P,Tibbits GF||Biophysical journal (93:2504)||2007|
Heterogeneous expression of Ca(2+) handling proteins in rabbit sinoatrial node.
MA3-926 was used in immunocytochemistry to study the densities of the L-type calcium current and proteins related to calcium handling in rabbit sinoatrial node
|Musa H,Lei M,Honjo H,Jones SA,Dobrzynski H,Lancaster MK,Takagishi Y,Henderson Z,Kodama I,Boyett MR||The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society (50:311)||2002|
Cardiac Resynchronization Therapy Reduces Subcellular Heterogeneity of Ryanodine Receptors, T-Tubules, and Ca2+ Sparks Produced by Dyssynchronous Heart Failure.
MA3-926 was used in immunocytochemistry to study dyssynchronous heart failure after cardiac resynchronization therapy
|Li H,Lichter JG,Seidel T,Tomaselli GF,Bridge JH,Sachse FB||Circulation. Heart failure (8:1105)||2015|
Progesterone modulates SERCA2a expression and function in rabbit cardiomyocytes.
MA3-926 was used in western blot to study the effect of progesterone on SERCA2a regulation in isolated cardiomyocytes
|Moshal KS,Zhang Z,Roder K,Kim TY,Cooper L,Patedakis Litvinov B,Lu Y,Reddy V,Terentyev D,Choi BR,Koren G||American journal of physiology. Cell physiology (307:C1050)||2014|
The topology of the C-terminal sections of the NCX1 Na (+) /Ca ( 2+) exchanger and the NCKX2 Na (+) /Ca ( 2+) -K (+) exchanger.
MA3-926 was used in western blot to study the topology the three C-terminal transmembrane segments of the NCX1 Na (+) /Ca ( 2+) exchanger and the NCKX2 Na (+) /Ca ( 2+) -K (+) exchanger
|Szerencsei RT,Kinjo TG,Schnetkamp PP||Channels (Austin, Tex.) (7:109)||2013|
Effects of SEA0400 on arrhythmogenicity in a Langendorff-perfused 1-month myocardial infarction rabbit model.
MA3-926 was used in western blot to study arrythomogenicity in a Langendorff-perfused rabbit heart model of myocardial infarction and the effects of a Na(+)/Ca(2+) exchanger inhibitor
|Chou CC,Chang PC,Wen MS,Lee HL,Chu Y,Baba A,Matsuda T,Yeh SJ,Wu D||Pacing and clinical electrophysiology : PACE (36:596)||2013|
Knockout of the Na,K-ATPase ¿2-isoform in cardiac myocytes delays pressure overload-induced cardiac dysfunction.
MA3-926 was used in western blot to study the role of cardiac myocyte Na,K-ATPase-alpha 2 in the devopment of cardiac dysfunction in response to pressure overload-induced
|Rindler TN,Lasko VM,Nieman ML,Okada M,Lorenz JN,Lingrel JB||American journal of physiology. Heart and circulatory physiology (304:H1147)||2013|
Bradycardia alters Ca(2+) dynamics enhancing dispersion of repolarization and arrhythmia risk.
MA3-926 was used in western blot to study the role of Ca(2+) metabolism in the mechanism by which bradycardia enhances dispersion of repolarization and increases the risk of arrhythmia
|Kim JJ,N¿mec J,Papp R,Strongin R,Abramson JJ,Salama G||American journal of physiology. Heart and circulatory physiology (304:H848)||2013|
Estradiol promotes sudden cardiac death in transgenic long QT type 2 rabbits while progesterone is protective.
MA3-926 was used in western blot to study the ability of estradiol to promote and progesterone to protect against sudden cardiac death in a rabbit model of long QT type 2
|Odening KE,Choi BR,Liu GX,Hartmann K,Ziv O,Chaves L,Schofield L,Centracchio J,Zehender M,Peng X,Brunner M,Koren G||Heart rhythm (9:823)||2012|
|Guinea pig||Not Cited||
NCX is an important determinant for premature ventricular activity in a drug-induced model of Andersen-Tawil syndrome.
MA3-926 was used in western blot to investigate the involvement of NCX in arrhythmias in a model of Andersen-Tawil syndrome
|Radwa¿ski PB,Poelzing S||Cardiovascular research (92:57)||2011|
Microvolt T-wave alternans and beat-to-beat variability of repolarization during early postischemic remodeling in a pig heart.
MA3-926 was used in western blot to study the interaction of T-wave alternans and beat-to-beat repolarisation variability in pig heart.
|Floré V,Claus P,Antoons G,Oosterhoff P,Holemans P,Vos MA,Sipido KR,Willems R||Heart rhythm (8:1050)||2011|
Baroreflex sensitivity impairment is associated with cardiac diastolic dysfunction in rats.
MA3-926 was used in western blot to investigate the relationship between baroreflex dysfunction and heart diastolic function in rodents
|Mostarda C,Moraes-Silva IC,Moreira ED,Medeiros A,Piratello AC,Consolim-Colombo FM,Caldini EG,Brum PC,Krieger EM,Irigoyen MC||Journal of cardiac failure (17:519)||2011|
Hyperglycemia can delay left ventricular dysfunction but not autonomic damage after myocardial infarction in rodents.
MA3-926 was used in western blot to identify the influence of hyperglycemia on ventricular function following myocardial infarction
|Rodrigues B,Rosa KT,Medeiros A,Schaan BD,Brum PC,De Angelis K,Irigoyen MC||Cardiovascular diabetology (10:null)||2011|
Angiotensin receptor blockade improves the net balance of cardiac Ca(2+) handling-related proteins in sympathetic hyperactivity-induced heart failure.
MA3-926 was used in western blot to study the effects on Ca(2+) metabolism of therapeutic blockade of angiotensin II type 1 receptors.
|Ferreira JC,Moreira JB,Campos JC,Pereira MG,Mattos KC,Coelho MA,Brum PC||Life sciences (88:578)||2011|
Decrease in the density of t-tubular L-type Ca2+ channel currents in failing ventricular myocytes.
MA3-926 was used in western blot to investigate the mechanism for the decrease of the TT LTCC current density in failing ventricular myocytes
|Horiuchi-Hirose M,Kashihara T,Nakada T,Kurebayashi N,Shimojo H,Shibazaki T,Sheng X,Yano S,Hirose M,Hongo M,Sakurai T,Moriizumi T,Ueda H,Yamada M||American journal of physiology. Heart and circulatory physiology (300:H978)||2011|
Discrepant electrophysiological characteristics and calcium homeostasis of left atrial anterior and posterior myocytes.
MA3-926 was used in western blot to investigate the difference between anterior and posterior myocytes in the left atrium
|Suenari K,Chen YC,Kao YH,Cheng CC,Lin YK,Chen YJ,Chen SA||Basic research in cardiology (106:65)||2011|
Aerobic exercise training improves skeletal muscle function and Ca2+ handling-related protein expression in sympathetic hyperactivity-induced heart failure.
MA3-926 was used in western blot to investigate the effect of aerobic exercise on skeletal muscle function and calcium-related protein expression
|Bueno CR,Ferreira JC,Pereira MG,Bacurau AV,Brum PC||Journal of applied physiology (Bethesda, Md. : 1985) (109:702)||2010|
Aerobic exercise training improves Ca2+ handling and redox status of skeletal muscle in mice.
MA3-926 was used in western blot to investigate the effect of aerobic exercise on calcium signal and redox state of skeletal muscle
|Ferreira JC,Bacurau AV,Bueno CR,Cunha TC,Tanaka LY,Jardim MA,Ramires PR,Brum PC||Experimental biology and medicine (Maywood, N.J.) (235:497)||2010|
Cardioprotection via modulation of calcium homeostasis by thiopental in hypoxia-reoxygenated neonatal rat cardiomyocytes.
MA3-926 was used in western blot to investigate the mechanism for the protective effect of thiopental against ischemia-reperfusion injury in rats
|Kim HS,Hwang KC,Park WK||Yonsei medical journal (51:187)||2010|
A transmural gradient in the cardiac Na/K pump generates a transmural gradient in Na/Ca exchange.
MA3-926 was used in western blot to investigate the relationship between cardiac Na/K pump and Na/Ca exchange
|Wang W,Gao J,Entcheva E,Cohen IS,Gordon C,Mathias RT||The Journal of membrane biology (233:51)||2010|
Enhanced calcium cycling and contractile function in transgenic hearts expressing constitutively active G alpha o* protein.
MA3-926 was used in western blot to investigate the role of G alpha o on calcium cycling and contractile function in heart
|Zhu M,Gach AA,Liu G,Xu X,Lim CC,Zhang JX,Mao L,Chuprun K,Koch WJ,Liao R,Koren G,Blaxall BC,Mende U||American journal of physiology. Heart and circulatory physiology (294:H1335)||2008|
Characterization of the phospholemman knockout mouse heart: depressed left ventricular function with increased Na-K-ATPase activity.
MA3-926 was used in western blot to study the functions of PLM in the contractile functions of the normoxic mouse heart
|Bell JR,Kennington E,Fuller W,Dighe K,Donoghue P,Clark JE,Jia LG,Tucker AL,Moorman JR,Marber MS,Eaton P,Dunn MJ,Shattock MJ||American journal of physiology. Heart and circulatory physiology (294:H613)||2008|
Thiol-oxidant monochloramine mobilizes intracellular Ca2+ in parietal cells of rabbit gastric glands.
MA3-926 was used in western blot to study the effect of monochloramine on intracellular Ca2+ mobilization in rabbit parietal cells
|Walsh BM,Naik HB,Dubach JM,Beshire M,Wieland AM,Soybel DI||American journal of physiology. Cell physiology (293:C1687)||2007|
SIK1 is part of a cell sodium-sensing network that regulates active sodium transport through a calcium-dependent process.
MA3-926 was used in western blot to study the role of SIK1 in intracellular sodium transport
|Sjöström M,Stenström K,Eneling K,Zwiller J,Katz AI,Takemori H,Bertorello AM||Proceedings of the National Academy of Sciences of the United States of America (104:16922)||2007|
Stress and high heart rate provoke ventricular tachycardia in mice expressing triadin.
MA3-926 was used in western blot to study the role of triadin in stress- and tachycardia-related ventricular arrhythmias
|Kirchhof P,Klimas J,Fabritz L,Zwiener M,Jones LR,Schäfers M,Hermann S,Boknik P,Schmitz W,Breithardt G,Kirchhefer U,Neumann J||Journal of molecular and cellular cardiology (42:962)||2007|
Disruption of the intracellular Ca2+ homeostasis in the cardiac excitation-contraction coupling is a crucial mechanism of arrhythmic toxicity in aconitine-induced cardiomyocytes.
MA3-926 was used in western blot to investigate the mechanism of arrhythmic cytotoxicity in heart cells treated with aconitine
|Fu M,Wu M,Wang JF,Qiao YJ,Wang Z||Biochemical and biophysical research communications (354:929)||2007|
Gene expression of the Na-Ca2+ exchanger, SERCA2a and calsequestrin after myocardial ischemia in the neonatal rabbit heart.
MA3-926 was used in western blot to investigate gene and protein expression of calcium handling proteins after myocardial ischemia in neonatal rabbit heart
|Seehase M,Quentin T,Wiludda E,Hellige G,Paul T,Schiffmann H||Biology of the neonate (90:174)||2006|
Distinct subcellular location of the Ca2+-binding protein S100A1 differentially modulates Ca2+-cycling in ventricular rat cardiomyocytes.
MA3-926 was used in western blot to investigate the role of distinct subcellular location of the calcium-binding protein S100A1 in cardiac calcium-handling
|Most P,Boerries M,Eicher C,Schweda C,Völkers M,Wedel T,Söllner S,Katus HA,Remppis A,Aebi U,Koch WJ,Schoenenberger CA||Journal of cell science (118:421)||2005|
Cardiac adenoviral S100A1 gene delivery rescues failing myocardium.
MA3-926 was used in western blot to investigaet the ability of S100A2 gene transfer to rescue failing myocardium
|Most P,Pleger ST,Völkers M,Heidt B,Boerries M,Weichenhan D,Löffler E,Janssen PM,Eckhart AD,Martini J,Williams ML,Katus HA,Remppis A,Koch WJ||The Journal of clinical investigation (114:1550)||2004|
Insights into cardioprotection obtained from study of cellular Ca2+ handling in myocardium of true hibernating mammals.
MA3-926 was used in western blot to study mammalian hibernators' inborn cardioprotective functions during hibernation
|Yatani A,Kim SJ,Kudej RK,Wang Q,Depre C,Irie K,Kranias EG,Vatner SF,Vatner DE||American journal of physiology. Heart and circulatory physiology (286:H2219)||2004|
Cellular mechanisms of contractile dysfunction in hibernating myocardium.
MA3-926 was used in western blot to investigate the phenotype of reduced contractility of the hibernating myocardium
|Bito V,Heinzel FR,Weidemann F,Dommke C,van der Velden J,Verbeken E,Claus P,Bijnens B,De Scheerder I,Stienen GJ,Sutherland GR,Sipido KR||Circulation research (94:794)||2004|
Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists.
MA3-926 was used in western blot to study the mechanism by which triadin 1 overexpression causes a blunted contractile response to beta-adrenergic agonists
|Kirchhefer U,Jones LR,Begrow F,Boknik P,Hein L,Lohse MJ,Riemann B,Schmitz W,Stypmann J,Neumann J||Cardiovascular research (62:122)||2004|
Na(+)-Ca(2+) exchanger overexpression predisposes to reactive oxygen species-induced injury.
MA3-926 was used in western blot to demonstrate that the overexpression of sodium/calcium exchanger is more susceptible to reactive oxygen species (ROS)-induced injury
|Wagner S,Seidler T,Picht E,Maier LS,Kazanski V,Teucher N,Schillinger W,Pieske B,Isenberg G,Hasenfuss G,Kögler H||Cardiovascular research (60:404)||2003|
Transgenic overexpression of the Ca2+-binding protein S100A1 in the heart leads to increased in vivo myocardial contractile performance.
MA3-926 was used in western blot to investigate the role of S100A1 in the regulation of cardiac function in vivo
|Most P,Remppis A,Pleger ST,Löffler E,Ehlermann P,Bernotat J,Kleuss C,Heierhorst J,Ruiz P,Witt H,Karczewski P,Mao L,Rockman HA,Duncan SJ,Katus HA,Koch WJ||The Journal of biological chemistry (278:33809)||2003|
Overexpression of Na+/Ca2+ exchanger gene attenuates postinfarction myocardial dysfunction.
MA3-926 was used in western blot to investigate the effects of overexpression of sodium/calcium exchanger gene on postinfarction myocardial dysfunction
|Min JY,Sullivan MF,Yan X,Feng X,Chu V,Wang JF,Amende I,Morgan JP,Philipson KD,Hampton TG||American journal of physiology. Heart and circulatory physiology (283:H2466)||2002|
Defective intracellular Ca(2+) signaling contributes to cardiomyopathy in Type 1 diabetic rats.
MA3-926 was used in western blot to study the possible contribution of defects in intracellular calcium signaling cardiomyopathy in streptozotocin (STZ)-induced diabetic rats
|Choi KM,Zhong Y,Hoit BD,Grupp IL,Hahn H,Dilly KW,Guatimosim S,Lederer WJ,Matlib MA||American journal of physiology. Heart and circulatory physiology (283:H1398)||2002|
Cardiac remodeling and atrial fibrillation in transgenic mice overexpressing junctin.
MA3-926 was used in western blot to study junctin's functions in heart
|Hong CS,Cho MC,Kwak YG,Song CH,Lee YH,Lim JS,Kwon YK,Chae SW,Kim DH||FASEB journal : official publication of the Federation of American Societies for Experimental Biology (16:1310)||2002|
Alkalosis stimulates endothelial nitric oxide synthase in cultured human pulmonary arterial endothelial cells.
MA3-926 was used in western blot to study the regulation of eNOS and NCX activity by pH and the corresponding mRNA aboundance and protein expression in HPAEC
|Mizuno S,Demura Y,Ameshima S,Okamura S,Miyamori I,Ishizaki T||American journal of physiology. Lung cellular and molecular physiology (283:L113)||2002|
Cardiac-specific IGF-1 expression attenuates dilated cardiomyopathy in tropomodulin-overexpressing transgenic mice.
MA3-926 was used in western blot to identify the factors related to the rescue of the TOT mouse from the development of a dilated myopathy and premature death by IGF-1
|Welch S,Plank D,Witt S,Glascock B,Schaefer E,Chimenti S,Andreoli AM,Limana F,Leri A,Kajstura J,Anversa P,Sussman MA||Circulation research (90:641)||2002|
Expressing and purifying membrane transport proteins in high yield.
MA3-926 was used in western blot to evaluate a protocol for the purification of membrane transport proteins
|Hale CC,Hill CK,Price EM,Bossuyt J||Journal of biochemical and biophysical methods (50:233)||2002|
Comparative analysis of the isoform expression pattern of Ca(2+)-regulatory membrane proteins in fast-twitch, slow-twitch, cardiac, neonatal and chronic low-frequency stimulated muscle fibers.
MA3-926 was used in western blot to study the expression of Ca(2+) regulatory membrane protein isoforms in skeletal, cardiac and neonatal muscle fibres
|Froemming GR,Murray BE,Harmon S,Pette D,Ohlendieck K||Biochimica et biophysica acta (1466:151)||2000|
Restoration of diastolic function in senescent rat hearts through adenoviral gene transfer of sarcoplasmic reticulum Ca(2+)-ATPase.
MA3-926 was used in western blot to investigate the possible relationship between the restoration of the SR calcium-ATPase through adenoviral gene transfer in the aging heart and the normalization of diastolic function in vivo
|Schmidt U,del Monte F,Miyamoto MI,Matsui T,Gwathmey JK,Rosenzweig A,Hajjar RJ||Circulation (101:790)||2000|
Effects of chronic low-frequency stimulation on Ca2+-regulatory membrane proteins in rabbit fast muscle.
MA3-926 was used in western blot to study the effects of chronic low frequency stimulation of fast-twitch muscle on the levels of Ca(2+) regulatory membrane proteins
|Ohlendieck K,Frömming GR,Murray BE,Maguire PB,Leisner E,Traub I,Pette D||Pflugers Archiv : European journal of physiology (438:700)||1999|
Mechanisms of altered excitation-contraction coupling in canine tachycardia-induced heart failure, I: experimental studies.
MA3-926 was used in western blot to demonstrate that the mechanisms of altered excitation-contraction coupling in canine are similar to those reported in human heart failure
|O'Rourke B,Kass DA,Tomaselli GF,Kääb S,Tunin R,Marbán E||Circulation research (84:562)||1999|
Colocalization of voltage-gated Na+ channels with the Na+/Ca2+ exchanger in rabbit cardiomyocytes during development.
MA3-926 was used in immunohistochemistry to investigate the localization of sodium homeostasis-related genes in rabbit heart cells
|Gershome C,Lin E,Kashihara H,Hove-Madsen L,Tibbits GF||American journal of physiology. Heart and circulatory physiology (300:H300)||2011|
Differential distribution of NCX1 contributes to spine-dendrite compartmentalization in CA1 pyramidal cells.
MA3-926 was used in immunohistochemistry to analyze the subcellular distribution of NCX1 and its role in calcium compartmentalization in rat CA1 pyramidal cells.
|Lörincz A,Rózsa B,Katona G,Vizi ES,Tamás G||Proceedings of the National Academy of Sciences of the United States of America (104:1033)||2007|
Intrarenal and cellular localization of CLC-K2 protein in the mouse kidney.
MA3-926 was used in immunohistochemistry to study the localization of the kidney specific CKC-K2 channel at the organ and cellular level in mouse
|Kobayashi K,Uchida S,Mizutani S,Sasaki S,Marumo F||Journal of the American Society of Nephrology : JASN (12:1327)||2001|
Adiponectin represses gluconeogenesis independent of insulin in hepatocytes.
MA3-926 was used in immunoprecipitation to investigate the effect of adiponectin on gluconeogenesis in hepatocytes and its mechanism
|Zhou H,Song X,Briggs M,Violand B,Salsgiver W,Gulve EA,Luo Y||Biochemical and biophysical research communications (338:793)||2005|
The cardiac Na+-Ca2+ exchanger binds to the cytoskeletal protein ankyrin.
MA3-926 was used in immunoprecipitation to investigate the interaction between cardiac sodium/calcium exchanger and ankyrin
|Li ZP,Burke EP,Frank JS,Bennett V,Philipson KD||The Journal of biological chemistry (268:11489)||1993|