PA1-028 was used in western blotting to expand our understanding of the mechanisms involved in Pb-induced mitochondrial apoptosis. Liu G, Wang ZK, Wang ZY et al. (2016) Mitochondrial permeability transition and its regulatory components are implicated in apoptosis of primary cultures of rat proximal tubular cells exposed to lead. Arch Toxicol 90(5): 1193–1209.
PA3-022 was used in western blotting to explore the effects of FK506-binding protein 51 (FKBP51) deletion on adipogenesis. Stechschulte LA, Hinds TD, Khuder SS et al. (2014) FKBP51 controls cellular adipogenesis through p38 kinase-mediated phosphorylation of GRa and PPARg. Mol Endocrinol 28: 1265–1275.
PA3-022 was used in western blotting to identify cyclophilin-40–interacting proteins and the cellular functions of cyclophilin-40. Park MS, Chu F, Xie J et al. (2011) Identification of cyclophilin-40-interacting proteins reveals potential cellular function of cyclophilin-40. Anal Biochem 410: 257–265.
PA1-028 was used in western blotting to investigate the effect of CypD on mitochondrial permeability regulation in liver and brain. Hansson MJ, Morota S, Chen L, et al. (2011) Cyclophilin D-sensitive mitochondrial permeability transition in adult human brain and liver mitochondria. J Neurotrauma 28: 143–153.
PA1-028 was used in western blotting to investigate the association between spatially distinct mitochondrial proteomes and dysfunction in the type 2 diabetic heart. Dabkowski ER, Baseler WA, Williamson CL et al. (2010) Mitochondrial dysfunction in the type 2 diabetic heart is associated with alterations in spatially distinct mitochondrial proteomes. Am J Physiol Heart Circ Physiol 299: H529–540.