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Citas y referencias (12)
Invitrogen™
Rhinohide™ Polyacrylamide Gel Strengthener Concentrate
El gel potenciador de poliacrilamida RhinoHide mejora la tecnología de los geles de poliacrilamida clásicos al fabricar geles mucho másMás información
| Número de catálogo | Cantidad |
|---|---|
| R33400 | 200 mL |
Número de catálogo R33400
Precio (CLP)
203.891
Each
Cantidad:
200 mL
Precio (CLP)
203.891
Each
El gel potenciador de poliacrilamida RhinoHide mejora la tecnología de los geles de poliacrilamida clásicos al fabricar geles mucho más fuertes, proporcionando un manejo más fácil y mayor resistencia al desgarro, sin efectos secundarios adversos. El gel potenciador de poliacrilamida RhinoHide está específicamente recomendado para geles de bajo porcentaje, geles de gran formato y geles sujetos a múltiples pasos de tinción y manipulación. El gel potenciador de poliacrilamida RhinoHide también está disponible en un kit (R-33410), que incluye una mezcla de acrilamida/bis-acrilamida (relación 37,5:1).
Para uso exclusivo en investigación. No apto para uso en procedimientos diagnósticos.
Especificaciones
Línea de productosRhinohide
Tipo de productoConcentrado de gel potenciador de poliacrilamida
Cantidad200 mL
Condiciones de envíoTemperatura ambiente
Unit SizeEach
Contenido y almacenamiento
Almacenar a temperatura ambiente.
Preguntas frecuentes
I am staining my protein gel with multiple stains over several days. How do I prevent excessive gel tearing?
Citations & References (12)
Citations & References
Abstract
Cytoskeleton/endoplasmic reticulum collapse induced by prostaglandin J2 parallels centrosomal deposition of ubiquitinated protein aggregates.
Journal:J Biol Chem
PubMed ID:16774923
'Many neurodegenerative disorders, such as Parkinson disease, exhibit inclusion bodies containing ubiquitinated proteins. The mechanisms implicated in this aberrant protein deposition remain elusive. In these disorders signs of inflammation are also apparent in the affected central nervous system areas. We show that prostaglandin J2 (PGJ2), an endogenous product of inflammation,
Regulation of class II beta-tubulin expression by tumor suppressor p53 protein in mouse melanoma cells in response to Vinca alkaloid.
Journal:Mol Cancer Res
PubMed ID:16603638
'The continuous exposure of antimicrotubule drugs to tumors often results in the emergence of drug-resistant tumor cells with altered expression of several beta-tubulin isotypes. We found that Vinca alkaloid enhanced expression of class II beta-tubulin isotype (mTUBB2) in mouse B16F10 melanoma cells via alteration of the tumor suppressor p53 protein.
Prostaglandin J2 alters pro-survival and pro-death gene expression patterns and 26 S proteasome assembly in human neuroblastoma cells.
Journal:J Biol Chem
PubMed ID:16737963
'Many neurodegenerative disorders are characterized by two pathological hallmarks: progressive loss of neurons and occurrence of inclusion bodies containing ubiquitinated proteins. Inflammation may be critical to neurodegeneration associated with ubiquitin-protein aggregates. We previously showed that prostaglandin J2 (PGJ2), one of the endogenous products of inflammation, induces neuronal death and the
Aging perturbs 26S proteasome assembly in Drosophila melanogaster.
Journal:FASEB J
PubMed ID:17413001
'Aging is associated with loss of quality control in protein turnover. The ubiquitin-proteasome pathway is critical to this quality control process as it degrades mutated and damaged proteins. We identified a unique aging-dependent mechanism that contributes to proteasome dysfunction in Drosophila melanogaster. Our studies are the first to show that
Proteomic analysis of a penicillin-tolerant rgg mutant strain of Streptococcus pyogenes.
Journal:J Antimicrob Chemother
PubMed ID:16891633
'To determine whether the transcriptional regulator Rgg contributes to penicillin-induced killing of Streptococcus pyogenes by altering a regulatory response to penicillin. Penicillin-induced killing of a wild-type and isogenic rgg mutant strain was assessed in broth and solid media and in the presence of cerulenin, which inhibits fatty acid biosynthesis (FAB).