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Citations & References (32)
Invitrogen™
QSY™ 7 Carboxylic Acid, Succinimidyl Ester
QSY™ 7 carboxylic acid, succinimidyl ester
| Catalog Number | Quantity |
|---|---|
| Q10193 also known as Q-10193 | 5 mg |
Catalog number Q10193
also known as Q-10193
Price (EUR)
550,00
Each
Quantity:
5 mg
Price (EUR)
550,00
Each
QSY-7 succinimidyl ester is a nonfluorescent acceptor dye for preparation of peptide and oligonucleotide FRET probes via aliphatic amine modification.
For Research Use Only. Not for use in diagnostic procedures.
Specifications
Chemical ReactivityAmine
Label or DyeQSY™ 7
Product TypeSuccinimidyl Ester
Quantity5 mg
Reactive MoietyActive Ester, Succinimidyl Ester
Shipping ConditionRoom Temperature
Label TypeQuencher Dyes
Product LineQSY
Unit SizeEach
Contents & Storage
Store in freezer (-5 to -30°C) and protect from light.
Citations & References (32)
Citations & References
Abstract
Identification and characterization of avian retroviruses in chicken embryo-derived yellow fever vaccines: investigation of transmission to vaccine recipients.
Journal:Journal of virology
PubMed ID:12502826
All currently licensed yellow fever (YF) vaccines are propagated in chicken embryos. Recent studies of chick cell-derived measles and mumps vaccines show evidence of two types of retrovirus particles, the endogenous avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the chicken embryonic fibroblast substrates. In
Genome-wide protective response used by group A Streptococcus to evade destruction by human polymorphonuclear leukocytes.
Journal:Proc Natl Acad Sci U S A
PubMed ID:12574517
Group A Streptococcus (GAS) evades polymorphonuclear leukocyte (PMN) phagocytosis and killing to cause human disease, including pharyngitis and necrotizing fasciitis (flesh-eating syndrome). We show that GAS genes differentially regulated during phagocytic interaction with human PMNs comprise a global pathogen-protective response to innate immunity. GAS prophage genes and genes involved in
Group A Streptococcus gene expression in humans and cynomolgus macaques with acute pharyngitis.
Journal:Infect Immun
PubMed ID:12654842
The molecular mechanisms used by group A Streptococcus (GAS) to survive on the host mucosal surface and cause acute pharyngitis are poorly understood. To provide new information about GAS host-pathogen interactions, we used real-time reverse transcription-PCR (RT-PCR) to analyze transcripts of 17 GAS genes in throat swab specimens taken from
Rapid diagnosis of Ebola hemorrhagic fever by reverse transcription-PCR in an outbreak setting and assessment of patient viral load as a predictor of outcome.
Journal:J Virol
PubMed ID:15047846
The largest outbreak on record of Ebola hemorrhagic fever (EHF) occurred in Uganda from August 2000 to January 2001. The outbreak was centered in the Gulu district of northern Uganda, with secondary transmission to other districts. After the initial diagnosis of Sudan ebolavirus by the National Institute for Virology in
The HtrA protease from Streptococcus pneumoniae digests both denatured proteins and the competence-stimulating peptide.
Journal:J Biol Chem
PubMed ID:23012372
'The HtrA protease of Streptococcus pneumoniae functions both in a general stress response role and as an error sensor that specifically represses genetic competence when the overall level of biosynthetic errors in cellular proteins is low. However, the mechanism through which HtrA inhibits development of competence has been unknown. We