Membrane Permeability Dead Cell Apoptosis Kit with PO-PRO™-1 and 7-Aminoactinomycin D, for flow cytometry
Citations & References (1)
Invitrogen™
Membrane Permeability Dead Cell Apoptosis Kit with PO-PRO™-1 and 7-Aminoactinomycin D, for flow cytometry
This flow cytometry product detects apoptosis on the basis of changes that occur in the permeability of cell membranes usingRead more
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Catalog Number
Quantity
V35123
1 kit
Catalog number V35123
Price (HKD)
4,137.00
1 kit
Add to cart
Quantity:
1 kit
Price (HKD)
4,137.00
1 kit
Add to cart
This flow cytometry product detects apoptosis on the basis of changes that occur in the permeability of cell membranes using PO-PRO™-1 and 7-aminoactinomycin D (7-AAD) nucleic acid stains. After staining a cell population with PO-PRO™-1 dye and 7-AAD, apoptotic cells show violet fluorescence, dead cells show violet and red fluorescence, and live cells show little or no fluorescence. These populations can easily be distinguished by a flow cytometer that uses both a violet laser and the 488 nm line of an argon-ion laser for excitation. Each kit contains sufficient reagents for approximately 200 flow cytometry tests.
For Use With (Equipment)Fluorescence Microscope, Flow Cytometer
No. of Reactions200
Product LineVybrant
Product TypeDead Cell Apoptosis Kit
Quantity1 kit
Shipping ConditionRoom Temperature
Conjugate7-AAD, PO-PRO-1
FormatTube, Slide
Unit Size1 kit
Contents & Storage
Contains 1 vial of PO-PRO™-1 dye (500 μL), and 1 vial of 7-AAD (7-aminoactinomycin D, 200 μL).
Store in refrigerator (2–8°C) and protect from light.
Citations & References (1)
Citations & References
Abstract
Panax notoginseng saponin is superior to aspirin in inhibiting platelet adhesion to injured endothelial cells through COX pathway in vitro.
Authors:Wang MM, Xue M, Xu YG, Miao Y, Kou N, Yang L, Zhang Y, Shi DZ
Journal:Thromb Res
PubMed ID:27043068
This study was designed to investigate the effect of Panax notoginseng saponin (PNS) on platelet adhesion to injured endothelial cells (ECs) and platelet activation induced by injured ECs, and to explore its underlying mechanisms. ... More