Attofluor™ Cell Chamber, for microscopy
Attofluor™ Cell Chamber, for microscopy
Invitrogen™

Attofluor™ Cell Chamber, for microscopy

The Attofluor cell chamber is a durable and practical coverslip holder designed for viewing live cell specimens on upright or深入閱讀
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產品號碼Quantity
A78163 Pack
產品號碼 A7816
價格 (TWD)
16,310.00
온라인 행사
Ends: 31-Dec-2025
23,300.00
您節省 6,990.00 (30%)
Each
新增至購物車
Quantity:
3 Pack
價格 (TWD)
16,310.00
온라인 행사
Ends: 31-Dec-2025
23,300.00
您節省 6,990.00 (30%)
Each
新增至購物車
The Attofluor cell chamber is a durable and practical coverslip holder designed for viewing live cell specimens on upright or inverted microscopes. Because the chamber is constructed from surgical stainless steel and is autoclavable, cells can be grown directly on coverslips within the unit. The O-ring seal design prevents samples from being contaminated by oil or leakage of media from the coverslips. The chamber accepts 25 mm round coverslips and mounts in a standard 35 mm diameter stage holder, and the 0.5 mm base dimension allows clearance for the objective when focusing. Spare O-rings for the Attofluor cell chamber are available in sets of ten (Cat. no. O-14804).
For Research Use Only. Not for use in diagnostic procedures.
規格
Diameter (Metric)35 mm
Quantity3 Pack
Shipping ConditionRoom Temperature
Product LineAttofluor
TypeImaging System Cell Chamber
Unit SizeEach
內容物與存放
Store at room temperature.

引用資料與參考文獻 (16)

引用資料與參考文獻
Abstract
Adenosine monophosphate-activated kinase alpha1 promotes endothelial barrier repair.
Authors:Creighton J, Jian M, Sayner S, Alexeyev M, Insel PA,
Journal:FASEB J
PubMed ID:21680893
'The vascular endothelium responds to damage through activation of multiple signaling events that restore cell-cell adhesion and vascular integrity. However, the molecular mechanisms that integrate these events are not clearly defined. Herein, we identify a previously unexpected role for adenosine monophosphate-activated protein kinase (AMPK) in pulmonary microvascular endothelial cell (PMVEC) ... More
Restriction of receptor movement alters cellular response: physical force sensing by EphA2.
Authors:Salaita K, Nair PM, Petit RS, Neve RM, Das D, Gray JW, Groves JT,
Journal:Science
PubMed ID:20223987
'Activation of the EphA2 receptor tyrosine kinase by ephrin-A1 ligands presented on apposed cell surfaces plays important roles in development and exhibits poorly understood functional alterations in cancer. We reconstituted this intermembrane signaling geometry between live EphA2-expressing human breast cancer cells and supported membranes displaying laterally mobile ephrin-A1. Receptor-ligand binding, ... More
Dynamics of flagellum- and pilus-mediated association of Pseudomonas aeruginosa with contact lens surfaces.
Authors:Tran VB, Fleiszig SM, Evans DJ, Radke CJ,
Journal:Appl Environ Microbiol
PubMed ID:21498762
'Flagella and pili are appendages that modulate attachment of Pseudomonas aeruginosa to solid surfaces. However, previous studies have mostly reported absolute attachment. Neither the dynamic roles of these appendages in surface association nor those of attachment phenotypes have been quantified. We used video microscopy to address this issue. Unworn, sterile, ... More
The histone deacetylase Hos2 forms an Hsp42-dependent cytoplasmic granule in quiescent yeast cells.
Authors:Liu IC, Chiu SW, Lee HY, Leu JY,
Journal:Mol Biol Cell
PubMed ID:22337769
'One of many physiological adjustments in quiescent cells is spatial regulation of specific proteins and RNA important for the entry to or exit from the stationary phase. By examining the localization of epigenetic-related proteins in Saccharomyces cerevisiae, we observed the formation of a reversible cytosolic ' ... More
Transmembrane water-flux through SLC4A11: a route defective in genetic corneal diseases.
Authors:Vilas GL, Loganathan SK, Liu J, Riau AK, Young JD, Mehta JS, Vithana EN, Casey JR,
Journal:
PubMed ID:23813972
'Three genetic corneal dystrophies [congenital hereditary endothelial dystrophy type 2 (CHED2), Harboyan syndrome and Fuchs endothelial corneal dystrophy] arise from mutations of the SLC4a11 gene, which cause blindness from fluid accumulation in the corneal stroma. Selective transmembrane water conductance controls cell size, renal fluid reabsorption and cell division. All known ... More