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Slc6a13 catalyzes sodium and chloride dependent transport of gamma-aminobutyric acid (GABA) to terminate GABA mediated synaptic activity. Slc6a13 is also a high-affinity gamma-aminobutyric acid (GABA) transporter, terminates biological actions of GABA by reuptake into cell, and its reuptake function utilizes sodium and chloride concentration gradients. Further, Slc6a13 may also aid in beta-alanine transport. Diseases associated with SLC6A13 include Traumatic Glaucoma and Neovascular Glaucoma.
100 µL
100 µg
100 µL
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