Lipopolysaccharides from <i>Salmonella</i> Minnesota, Alexa Fluor™ 488 conjugate

Citations et références (6)

Invitrogen™

Lipopolysaccharides from Salmonella Minnesota, Alexa Fluor™ 488 conjugate

Ce conjugué brillant et vert fluorescent Alexa Fluor 488 peut être utilisé pour suivre les processus de liaison, de transportAfficher plus

Référence	Quantité
L23356	100 μg

Référence L23356

Prix (EUR)

397,00

Quantité:

100 μg

Prix (EUR)

397,00

Ce conjugué brillant et vert fluorescent Alexa Fluor 488 peut être utilisé pour suivre les processus de liaison, de transport et d’internalisation des cellules lipopolysaccharides.

Usage exclusivement réservé à la recherche. Ne pas utiliser pour des procédures de diagnostic.

Spécifications

Méthode de détectionFluorescence

Type de colorantColorants Alexa Fluor

Excitation / émission495/519

Quantité100 μg

Conditions d’expéditionTempérature ambiante

Gamme de produitsAlexa Fluor

Type de produitConjugué lipopolysaccharide

Unit SizeEach

Contenu et stockage

Stocker au congélateur (entre -5 et -30°C) à l’abri de la lumière.

Citations et références (6)

Citations et références

Abstract

Surfactant protein A directly interacts with TLR4 and MD-2 and regulates inflammatory cellular response. Importance of supratrimeric oligomerization.

Authors:Yamada C, Sano H, Shimizu T, Mitsuzawa H, Nishitani C, Himi T, Kuroki Y

Journal:J Biol Chem

PubMed ID:16754682

'The purpose of the current study was to examine the binding of pulmonary surfactant protein A (SP-A) to TLR4 and MD-2, which are critical signaling receptors for lipopolysaccharides (LPSs). The direct binding of SP-A to the recombinant soluble form of extracellular TLR4 domain (sTLR4) and MD-2 was detected using solid-phase ... More

Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway.

Authors:Lehnardt S, Massillon L, Follett P, Jensen FE, Ratan R, Rosenberg PA, Volpe JJ, Vartanian T

Journal:Proc Natl Acad Sci U S A

PubMed ID:12824464

Innate immunity is an evolutionarily ancient system that provides organisms with immediately available defense mechanisms through recognition of pathogen-associated molecular patterns. We show that in the CNS, specific activation of innate immunity through a Toll-like receptor 4 (TLR4)-dependent pathway leads to neurodegeneration. We identify microglia as the major lipopolysaccharide (LPS)-responsive ... More

Lipopolysaccharide (LPS) labeled with Alexa 488 hydrazide as a novel probe for LPS binding studies.

Authors:Triantafilou K, Triantafilou M, Fernandez N

Journal:Cytometry

PubMed ID:11084617

BACKGROUND: Lipopolysaccharide (LPS) comprises the outer cell wall of all gram-negative bacteria. It consists of an oligosaccharide core and lipid A. All LPS-induced biological responses are lipid A-dependent. Once released, LPS triggers a host systemic inflammatory response that leads to septic shock. Binding studies have helped to reveal some of ... More

The toll-like receptor TLR4 is necessary for lipopolysaccharide-induced oligodendrocyte injury in the CNS.

Authors:Lehnardt S, Lachance C, Patrizi S, Lefebvre S, Follett PL, Jensen FE, Rosenberg PA, Volpe JJ, Vartanian T

Journal:J Neurosci

PubMed ID:11923412

The immediate or innate immune response is the first line of defense against diverse microbial pathogens and requires the expression of recently discovered toll-like receptors (TLRs). TLR4 serves as a specific receptor for lipopolysaccharide (LPS) and is localized on the surface of a subset of mammalian cells. Although innate immunity ... More

Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.

Authors:

Journal:PLoS One

PubMed ID:27171436

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