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          System Message

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          LOADING ...
          Gene:
          PRMT2
          Transcript:
          ENST00000397637.0
          Assay Name:
          Mutation genome location:
          chr.21 46648380-46648879 on build GRCh38
          Assay ID Hs00361026_CE

          Purification
          Size
          Availability Made To Order
          Catalog # A15629
          Price
          Your Price
          Online offer:
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          • Genomic Map
          • Assay Details
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          Genomic Map

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          Assay Details


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          More Information


          Set Membership:

          Between 400-700 bp

          Panther Classification:

          Molecular Function -

          protein modifying enzyme

          Gene Ontology Categories:

          Function(s) Process(es)

          protein methylation
          signal transduction
          histone methylation
          peptidyl-arginine methylation, to asymmetrical-dimethyl arginine
          negative regulation of NF-kappaB transcription factor activity
          histone arginine methylation
          positive regulation of apoptotic process
          negative regulation of transcription, DNA-templated
          positive regulation of transcription, DNA-templated
          developmental cell growth
          regulation of androgen receptor signaling pathway
          negative regulation of G1/S transition of mitotic cell cycle
          transcription coactivator activity
          signal transducer activity
          protein binding
          histone-arginine N-methyltransferase activity
          protein-arginine N-methyltransferase activity
          estrogen receptor binding
          progesterone receptor binding
          protein-arginine omega-N asymmetric methyltransferase activity
          histone methyltransferase activity
          protein homodimerization activity
          retinoic acid receptor binding
          peroxisome proliferator activated receptor binding
          thyroid hormone receptor binding
          androgen receptor binding

          Back To Top

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