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Alpha-Bungarotoxin Conjugates
Alpha-Bungarotoxin Conjugates
Citations & References (13)
Invitrogen™

Alpha-Bungarotoxin Conjugates

Thermo Fisher Scientific offers a broad selection of Invitrogen alpha-bungarotoxin conjugates, including Alexa Fluor and Alexa Fluor Plus conjugates.
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製品番号（カタログ番号）標識または色素
B35451Alexa Fluor色素
B56130Alexa Fluor™ Plus 405
B13422Alexa Fluor色素
B13423Alexa Fluor™ 594
B35450Alexa Fluor色素
B1196ビオチン＆その他のハプテン
T1175Tetramethylrhodamine
B1601非標識
製品番号（カタログ番号） B35451
価格（JPY）
89,700
Online offer
Ends: 27-Mar-2026
128,200
割引額 38,500 (30%)
Each
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標識または色素:
Alexa Fluor色素
Thermo Fisher Scientific offers a broad selection of Invitrogen alpha-bungarotoxin conjugates, including Alexa Fluor and Alexa Fluor Plus conjugates. Alpha-bungarotoxin, a 74-amino acid peptide extracted from Bungarus multicinctus venom, binds with high affinity to the alpha subunit of the nicotinic acetylcholine receptor (AChR) of neuromuscular junctions.

Our bright and photostable Alexa Fluor 555 alpha-bungarotoxin is a superior choice of probe for visualizing this receptor. Fluorescent alpha-bungarotoxin conjugates can be used to facilitate identification of nicotinic AChRs and localization of neuromuscular junctions. Conjugated alpha-bungarotoxin has been used in variety of applications, including immunofluorescence (IF), immunohistochemistry (IHC), and flow cytometry (FC).

For Research Use Only. Not for use in diagnostic procedures.
仕様
標識または色素Alexa Fluor色素
製品タイプコンジュゲート
タンパク質サブタイプその他のタンパク質
数量500 μg
出荷条件室温
Excitation/Emission555/565 nm
製品ラインAlexa Fluor
Unit SizeEach
組成および保存条件
フリーザー（-5～-30度）に保存し、遮光してください。

引用および参考文献 (13)

引用および参考文献
Abstract
Agrin elicits membrane lipid condensation at sites of acetylcholine receptor clusters in C2C12 myotubes.
Authors:Stetzkowski-Marden F, Gaus K, Recouvreur M, Cartaud A, Cartaud J
Journal:J Lipid Res
PubMed ID:16816402
'The formation of the neuromuscular junction is characterized by the progressive accumulation of nicotinic acetylcholine receptors (AChRs) in the postsynaptic membrane facing the nerve terminal, induced predominantly through the agrin/muscle-specific kinase (MuSK) signaling cascade. However, the cellular mechanisms linking MuSK activation to AChR clustering are still poorly understood. Here, we ... More
Impaired synaptic vesicle release and immaturity of neuromuscular junctions in spinal muscular atrophy mice.
Authors:Kong L, Wang X, Choe DW, Polley M, Burnett BG, Bosch-Marcé M, Griffin JW, Rich MM, Sumner CJ,
Journal:J Neurosci
PubMed ID:19158308
'The motor neuron disease spinal muscular atrophy (SMA) causes profound muscle weakness that most often leads to early death. At autopsy, SMA is characterized by loss of motor neurons and muscle atrophy, but the initial cellular events that precipitate motor unit dysfunction and loss remain poorly characterized. Here, we examined ... More
Neuromuscular junction integrity after chronic nerve compression injury.
Authors:Mozaffar T, Strandberg E, Abe K, Hilgenberg LG, Smith MA, Gupta R,
Journal:J Orthop Res
PubMed ID:18655131
Chronic nerve compression injuries (CNC) are progressive demyelinating disorders characterized by a gradual decline of the nerve conduction velocity (NCV) in the affected nerve region. CNC injury induces a robust Schwann cell response with axonal sprouting, but without morphologic evidence of axonal injury. We hypothesize that early CNC injury occurs ... More
CXCL12α/SDF-1 from perisynaptic Schwann cells promotes regeneration of injured motor axon terminals.
Authors:
Journal:EMBO Mol Med
PubMed ID:28559442
Spinal subpial delivery of AAV9 enables widespread gene silencing and blocks motoneuron degeneration in ALS.
Authors:
Journal:Nat Med
PubMed ID:31873312
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