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Citations & References (6)
Invitrogen™
Lipopolysaccharides from Salmonella Minnesota, Alexa Fluor™ 488 conjugate
この明るい緑色蛍光のAlexa Fluor 488コンジュゲートは、リポ多糖の結合、輸送、細胞内在化のプロセスを追跡するために使用できます。詳細を見る
| 製品番号（カタログ番号） | 数量 |
|---|---|
| L23356 | 100 μg |
製品番号（カタログ番号） L23356
価格（JPY）
33,000
Online offer
Ends: 27-Mar-2026
55,000割引額 22,000 (40%)
Each
数量:
100 μg
この明るい緑色蛍光のAlexa Fluor 488コンジュゲートは、リポ多糖の結合、輸送、細胞内在化のプロセスを追跡するために使用できます。
研究用にのみ使用できます。診断用には使用いただけません。
仕様
検出法蛍光
染色剤タイプAlexa Fluor Dyes
励起／発光495⁄519
数量100 μg
出荷条件室温
製品ラインAlexa Fluor
製品タイプリポ多糖類コンジュゲート
Unit SizeEach
組成および保存条件
フリーザー（-5～-30度）に保存し、遮光してください。
引用および参考文献 (6)
引用および参考文献
Abstract
Surfactant protein A directly interacts with TLR4 and MD-2 and regulates inflammatory cellular response. Importance of supratrimeric oligomerization.
Journal:J Biol Chem
PubMed ID:16754682
'The purpose of the current study was to examine the binding of pulmonary surfactant protein A (SP-A) to TLR4 and MD-2, which are critical signaling receptors for lipopolysaccharides (LPSs). The direct binding of SP-A to the recombinant soluble form of extracellular TLR4 domain (sTLR4) and MD-2 was detected using solid-phase
Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway.
Journal:Proc Natl Acad Sci U S A
PubMed ID:12824464
Innate immunity is an evolutionarily ancient system that provides organisms with immediately available defense mechanisms through recognition of pathogen-associated molecular patterns. We show that in the CNS, specific activation of innate immunity through a Toll-like receptor 4 (TLR4)-dependent pathway leads to neurodegeneration. We identify microglia as the major lipopolysaccharide (LPS)-responsive
Lipopolysaccharide (LPS) labeled with Alexa 488 hydrazide as a novel probe for LPS binding studies.
Journal:Cytometry
PubMed ID:11084617
BACKGROUND: Lipopolysaccharide (LPS) comprises the outer cell wall of all gram-negative bacteria. It consists of an oligosaccharide core and lipid A. All LPS-induced biological responses are lipid A-dependent. Once released, LPS triggers a host systemic inflammatory response that leads to septic shock. Binding studies have helped to reveal some of
The toll-like receptor TLR4 is necessary for lipopolysaccharide-induced oligodendrocyte injury in the CNS.
Journal:J Neurosci
PubMed ID:11923412
The immediate or innate immune response is the first line of defense against diverse microbial pathogens and requires the expression of recently discovered toll-like receptors (TLRs). TLR4 serves as a specific receptor for lipopolysaccharide (LPS) and is localized on the surface of a subset of mammalian cells. Although innate immunity
Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.
Journal:PLoS One
PubMed ID:27171436