이 새로운 7500 Real-Time PCR System용 21 CFR Part 11 Module은 사용자가 설정할 수 있는 활용도 높은 CFR 구성 설정을자세히 알아보기
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카탈로그 번호
수량
4377354
1 kit
카탈로그 번호 4377354
제품 가격(KRW)
7,460,000
Each
카트에 추가하기
수량:
1 kit
제품 가격(KRW)
7,460,000
Each
카트에 추가하기
이 새로운 7500 Real-Time PCR System용 21 CFR Part 11 Module은 사용자가 설정할 수 있는 활용도 높은 CFR 구성 설정을 제공하여 연방규정집(CRF) Title 21 Part 11 준수를 돕습니다.
연구용으로만 사용할 수 있습니다. 진단 절차에 사용할 수 없습니다.
For Research Use Only. Not for use in diagnostic procedures.
사양
깊이(영국식 단위)17.72 in.
깊이(미터법)45 cm
용도(애플리케이션)Real Time PCR (qPCR)
용도(장비)7500 System
높이(영국식 단위)19.29 in.
높이(미터법)49 cm
수량1 kit
소프트웨어 유형Software
열 정확도±0.0.25°C
열 범위4°C to 100°C
열 균일성±0.0°C (30 s after Clock Start)
워런티1 Year
무게34 kg (75 lb.)
폭(영국식 단위)13.99 in.
폭(미터법)34 cm
블럭 구성96-well
블록 형식Peltier-based
용량96-well Plate x 0.2 mL Tubes
제품 유형7500 System SDS
Unit SizeEach
자주 묻는 질문(FAQ)
Are there differences in the tube/plate format between Applied Biosystems 7500 Real-Time PCR Systems and Applied Biosystems 7500 Fast Real-Time PCR Systems?
Yes, the standard 7500 instrument has a 96 x 0.2 mL block, whereas the 7500 Fast instrument has a 96 x 0.1 mL block.
Role of APE1 in differentiated neuroblastoma SH-SY5Y cells in response to oxidative stress: Use of APE1 small molecule inhibitors to delineate APE1 functions
Oxidative DNA damage has been implicated in a number of central nervous system pathologies. The base excision repair (BER) pathway is one of the most important cellular protection mechanisms that respond to oxidative DNA damage. Human apurinic (apyrimidinic) endonuclease/redox effector factor (APE1/Ref1 or APE1) is an essential enzyme in the ... More
Characterization of matrix metalloproteinase-2 and matrix metalloproteinase-9 and their inhibitors in equine granulosa cells in vivo and in vitro
Authors:Sessions, DR; Vick, MM; Fitzgerald, BP
Journal:JOURNAL OF ANIMAL SCIENCE
PubMed ID:
Matrix metalloproteinases (MMP) and tissue inhibitors of MMP (TIMP) regulate tissue remodeling events necessary for ovulation. Thus, changes in MMP and TIMP expression and protein enzyme activity were examined in vivo and in vitro during follicular development and atresia in the horse. Equine granulosa cells and follicular fluid from medium ... More
The time of prenatal immune challenge determines the specificity of inflammation-mediated brain and behavioral pathology.
Authors:Meyer U, Nyffeler M, Engler A, Urwyler A, Schedlowski M, Knuesel I, Yee BK, Feldon J
Journal:J Neurosci
PubMed ID:16672647
Disturbance to early brain development is implicated in several neuropsychiatric disorders including autism, schizophrenia, and mental retardation. Epidemiological studies have indicated that the risk of developing these disorders is enhanced by prenatal maternal infection, presumably as a result of neurodevelopmental defects triggered by cytokine-related inflammatory events. Here, we demonstrate that ... More
Distinct roles for IFN regulatory factor (IRF)-3 and IRF-7 in the activation of antitumor properties of human macrophages.
Authors:Romieu-Mourez R, Solis M, Nardin A, Goubau D, Baron-Bodo V, Lin R, Massie B, Salcedo M, Hiscott J
Journal:Cancer Res
PubMed ID:17079482
When properly activated, macrophages can be tumoricidal, thus making them attractive additions to standard cancer therapies. To this end, tolerance and activity of human autologous IFN-gamma-activated macrophages, produced in large scale for clinical use (MAK cells), have been assessed in pilot trials in cancer patients. In the present study, we ... More
Trps1 plays a pivotal role downstream of Gdf5 signaling in promoting chondrogenesis and apoptosis of ATDC5 cells.
Authors:Itoh S, Kanno S, Gai Z, Suemoto H, Kawakatsu M, Tanishima H, Morimoto Y, Nishioka K, Hatamura I, Yoshida M, Muragaki Y
Journal:Genes Cells
PubMed ID:18363966
Tricho-rhino-phalangeal syndrome (TRPS) is an autosomal dominant skeletal disorder caused by mutations of TRPS1. Based on the similar expression patterns of Trps1 and Gdf5, we hypothesized a possible functional interaction between these two molecules. Using a chondrogenic cell line (ATDC5), we investigated the association of Gdf5-mediated signaling pathways with ... More