Product References
TET1s deficiency exacerbates oscillatory shear flow-induced atherosclerosis.
International journal of biological sciences
Qu K,Wang C,Huang L,Qin X,Zhang K,Zhong Y,Ma Q,Yan W,Li T,Peng Q,Wang Y,Gregersen H,Tang C,Qiu J,Wang G
Published figure using Connexin 40 polyclonal antibody (Product # 36-4900) in Western Blot
Fri Apr 15 00:00:00 EDT 2022
Atrial Fibrillation in Heart Failure Is Associated with High Levels of Circulating microRNA-199a-5p and 22-5p and a Defective Regulation of Intracellular Calcium and Cell-to-Cell Communication.
International journal of molecular sciences
Garcia-Elias A,Tajes M,Yañez-Bisbe L,Enjuanes C,Comín-Colet J,Serra SA,Fernández-Fernández JM,Aguilar-Agon KW,Reilly S,Martí-Almor J,Benito B
36-4900 was used in Western Blot to show that circulating miR-199a-5p and miR-22-5p are higher in heart failure with reduced ejection fraction patients with atrial fibrillation (AF), and that cells exposed to both miRNAs exhibited altered Ca2+ handling and defective cell-to-cell communication.
Sun Sep 26 00:00:00 EDT 2021
Atrial Fibrillation in Heart Failure Is Associated with High Levels of Circulating microRNA-199a-5p and 22-5p and a Defective Regulation of Intracellular Calcium and Cell-to-Cell Communication.
International journal of molecular sciences
Garcia-Elias A,Tajes M,Yañez-Bisbe L,Enjuanes C,Comín-Colet J,Serra SA,Fernández-Fernández JM,Aguilar-Agon KW,Reilly S,Martí-Almor J,Benito B
36-4900 was used in Western Blot to show that circulating miR-199a-5p and miR-22-5p are higher in heart failure with reduced ejection fraction patients with atrial fibrillation (AF), and that cells exposed to both miRNAs exhibited altered Ca2+ handling and defective cell-to-cell communication.
Sun Sep 26 00:00:00 EDT 2021
Role of atrial arrhythmia and ventricular response in atrial fibrillation induced atrial remodelling.
Cardiovascular research
Guichard JB,Xiong F,Qi XY,L'Heureux N,Hiram R,Xiao J,Naud P,Tardif JC,Da Costa A,Nattel S
36-4900 was used in Western Blotting to assess the roles of atrial arrhythmia versus high ventricular rate in AF-associated remodeling.
Thu Jan 21 00:00:00 EST 2021
Importance of endothelial Hey1 expression for thoracic great vessel development and its distal enhancer for Notch-dependent endothelial transcription.
The Journal of biological chemistry
Watanabe Y,Seya D,Ihara D,Ishii S,Uemoto T,Kubo A,Arai Y,Isomoto Y,Nakano A,Abe T,Shigeta M,Kawamura T,Saito Y,Ogura T,Nakagawa O
Published figure using Connexin 40 polyclonal antibody (Product # 36-4900) in Immunohistochemistry
Fri Dec 18 00:00:00 EST 2020
Vascular endothelial growth factor promotes atrial arrhythmias by inducing acute intercalated disk remodeling.
Scientific reports
Mezache L,Struckman HL,Greer-Short A,Baine S,Györke S,Radwański PB,Hund TJ,Veeraraghavan R
36-4900 was used in Western Blotting to show VEGF can slow atrial conduction and predispose normal hearts to atrial arrhythmias.
Tue Nov 24 00:00:00 EST 2020
Vascular endothelial growth factor promotes atrial arrhythmias by inducing acute intercalated disk remodeling.
Scientific reports
Mezache L,Struckman HL,Greer-Short A,Baine S,Györke S,Radwański PB,Hund TJ,Veeraraghavan R
36-4900 was used in Western Blotting to show VEGF can slow atrial conduction and predispose normal hearts to atrial arrhythmias.
Tue Nov 24 00:00:00 EST 2020
Systemic therapy in an RNA toxicity mouse model with an antisense oligonucleotide therapy targeting a non-CUG sequence within the DMPK 3'UTR RNA.
Human molecular genetics
Yadava RS,Yu Q,Mandal M,Rigo F,Bennett CF,Mahadevan MS
36-4900 was used in Immunohistochemistry-immunofluorescence to demonstrate systemic treatment with an antisense oligonucleotide (ASO) (ISIS 486178) targeted to a non-CUG sequence within the 3'UTR of dystrophia myotonica protein kinase.
Wed Jun 03 00:00:00 EDT 2020
An N-/L-type calcium channel blocker, cilnidipine, suppresses autonomic, electrical, and structural remodelling associated with atrial fibrillation.
Cardiovascular research
Tajiri K,Guichard JB,Qi X,Xiong F,Naud P,Tardif JC,Costa AD,Aonuma K,Nattel S
36-4900 was used in Western Blotting to investigate the effects of Cilnidipine, a commercially available drug with NTCC-blocking action, on atrial fibrillation-related remodelling.
Sun Dec 01 00:00:00 EST 2019
Implantation of engineered conduction tissue in the rat heart.
Molecular medicine reports
Zhang W,Li X,Sun S,Zhang X
36-4900 was used in Immunohistochemistry-immunofluorescence to determine whether engineered conduction tissue could act as an atrioventricular conduction pathway.
Mon Apr 01 00:00:00 EDT 2019
Shear-induced Notch-Cx37-p27 axis arrests endothelial cell cycle to enable arterial specification.
Nature communications
Fang JS,Coon BG,Gillis N,Chen Z,Qiu J,Chittenden TW,Burt JM,Schwartz MA,Hirschi KK
36-4900 was used in Immunocytochemistry to elucidate a mechanochemical pathway where arterial shear activates a NOTCH-GJA4-CDKN1B axis promoting endothelial cell cycle arrest to enable arterial gene expression.
Fri Dec 15 00:00:00 EST 2017
Mitochondrial connexin40 regulates mitochondrial calcium uptake in coronary endothelial cells.
American journal of physiology. Cell physiology
Guo R,Si R,Scott BT,Makino A
364900 was used in immunocytochemistry to determine the role of mitochondrial Cx40 in endothelial cells
Sat Apr 01 00:00:00 EDT 2017
Intermittent hypoxia causes NOX2-dependent remodeling of atrial connexins.
BMC cell biology
Gemel J,Su Z,Gileles-Hillel A,Khalyfa A,Gozal D,Beyer EC
36-4900 was used in immunohistochemistry and western blot to examine connexin40 and connexin43 expression in mouse models of hypoxia and sleep apnea
Tue Jan 17 00:00:00 EST 2017
Specificity of the connexin W3/4 locus for functional gap junction formation.
Channels (Austin, Tex.)
Xu Q,Lin X,Matiukas A,Zhang X,Veenstra RD
36-4900 was used in Immunocytochemistry to indicate an absolute requirement for an amphipathic W3/4 residue that is conserved among all α/Β/δ/γ-group connexins.
Tue Nov 01 00:00:00 EDT 2016
Autonomous beating rate adaptation in human stem cell-derived cardiomyocytes.
Nature communications
Eng G,Lee BW,Protas L,Gagliardi M,Brown K,Kass RS,Keller G,Robinson RB,Vunjak-Novakovic G
36-4900 was used in immunocytochemistry to determine the autonomouse beating rate adaptation in cardiomyocytes from human stem cells
Tue Jan 19 00:00:00 EST 2016
Functional formation of heterotypic gap junction channels by connexins-40 and -43.
Channels (Austin, Tex.)
Lin X,Xu Q,Veenstra RD
36-4900 was used in Western Blotting to examine the formation and biophysical properties of heterotypic Connexin43/Connexin40 gap junction channels.
Tue Sep 29 00:00:00 EDT 2015
Functional formation of heterotypic gap junction channels by connexins-40 and -43.
Channels (Austin, Tex.)
Lin X,Xu Q,Veenstra RD
36-4900 was used in Western Blotting to examine the formation and biophysical properties of heterotypic Connexin43/Connexin40 gap junction channels.
Tue Sep 29 00:00:00 EDT 2015
MMP-9- and NMDA receptor-mediated mechanism of diabetic renovascular remodeling and kidney dysfunction: hydrogen sulfide is a key modulator.
Nitric oxide : biology and chemistry
Kundu S,Pushpakumar S,Sen U
36-4900 was used in western blot to test if H2S supplementation reduces MMP-9-induced extracellular matrix remodeling and dysfunction in diabetic kidney.
Thu Apr 30 00:00:00 EDT 2015
Connexin40 abnormalities and atrial fibrillation in the human heart.
Journal of molecular and cellular cardiology
Gemel J,Levy AE,Simon AR,Bennett KB,Ai X,Akhter S,Beyer EC
Sat Nov 01 00:00:00 EDT 2014
Connexin40 abnormalities and atrial fibrillation in the human heart.
Journal of molecular and cellular cardiology
Gemel J,Levy AE,Simon AR,Bennett KB,Ai X,Akhter S,Beyer EC
Sat Nov 01 00:00:00 EDT 2014
Degradation of a connexin40 mutant linked to atrial fibrillation is accelerated.
Journal of molecular and cellular cardiology
Gemel J,Simon AR,Patel D,Xu Q,Matiukas A,Veenstra RD,Beyer EC
36-4900 was used in western blot to study Cx40 mutants and determine how they contribute to atrial fibrillation pathogenesis
Mon Sep 01 00:00:00 EDT 2014
Degradation of a connexin40 mutant linked to atrial fibrillation is accelerated.
Journal of molecular and cellular cardiology
Gemel J,Simon AR,Patel D,Xu Q,Matiukas A,Veenstra RD,Beyer EC
36-4900 was used in western blot to study Cx40 mutants and determine how they contribute to atrial fibrillation pathogenesis
Mon Sep 01 00:00:00 EDT 2014
Degradation of a connexin40 mutant linked to atrial fibrillation is accelerated.
Journal of molecular and cellular cardiology
Gemel J,Simon AR,Patel D,Xu Q,Matiukas A,Veenstra RD,Beyer EC
36-4900 was used in western blot to study Cx40 mutants and determine how they contribute to atrial fibrillation pathogenesis
Mon Sep 01 00:00:00 EDT 2014
Low density lipoproteins promote unstable calcium handling accompanied by reduced SERCA2 and connexin-40 expression in cardiomyocytes.
PloS one
Barriga M,Cal R,Cabello N,Llach A,Vallmitjana A,Benítez R,Badimon L,Cinca J,Llorente-Cortés V,Hove-Madsen L
36-4900 was used in western blot to test if low density lipoprotein cholesterol affects calcium dynamics and signal propagation in cultured atrial myocytes.
Thu Sep 12 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Autosomal recessive GJA1 (Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms.
Journal of cell science
Huang T,Shao Q,MacDonald A,Xin L,Lorentz R,Bai D,Laird DW
36-4900 was used in western blot to study two autosomal recessive mutations reported that cause oculodentodigital dysplasia.
Mon Jul 01 00:00:00 EDT 2013
Epistatic rescue of Nkx2.5 adult cardiac conduction disease phenotypes by prospero-related homeobox protein 1 and HDAC3.
Circulation research
Risebro CA,Petchey LK,Smart N,Gomes J,Clark J,Vieira JM,Yanni J,Dobrzynski H,Davidson S,Zuberi Z,Tinker A,Shui B,Tallini YI,Kotlikoff MI,Miquerol L,Schwartz RJ,Riley PR
36-4900 was used in Immunohistochemistry-immunofluorescence to identify bone fide upstream genetic modifiers of Nkx2.5/NKX2.5 function, and determine epistatic effects relevant to the manifestation of NKX2.5-dependent adult congenital heart disease.
Fri Jul 06 00:00:00 EDT 2012
TBX5 drives Scn5a expression to regulate cardiac conduction system function.
The Journal of clinical investigation
Arnolds DE,Liu F,Fahrenbach JP,Kim GH,Schillinger KJ,Smemo S,McNally EM,Nobrega MA,Patel VV,Moskowitz IP
36-4900 was used in immunohistochemistry to test if TBX5 regulates transcriptional networks required for mature cardiac conduction system function.
Sun Jul 01 00:00:00 EDT 2012
TBX5 drives Scn5a expression to regulate cardiac conduction system function.
The Journal of clinical investigation
Arnolds DE,Liu F,Fahrenbach JP,Kim GH,Schillinger KJ,Smemo S,McNally EM,Nobrega MA,Patel VV,Moskowitz IP
36-4900 was used in immunohistochemistry to test if TBX5 regulates transcriptional networks required for mature cardiac conduction system function.
Sun Jul 01 00:00:00 EDT 2012
Remodeling of the peripheral cardiac conduction system in response to pressure overload.
American journal of physiology. Heart and circulatory physiology
Harris BS,Baicu CF,Haghshenas N,Kasiganesan H,Scholz D,Rackley MS,Miquerol L,Gros D,Mukherjee R,O'Brien TX
36-4900 was used in immunohistochemistry to assess how chronic pressure overload affects the Purkinje fibers of the ventricular peripheral conduction system.
Sun Apr 15 00:00:00 EDT 2012
Inducible recombination in the cardiac conduction system of minK: CreERT² BAC transgenic mice.
Genesis (New York, N.Y. : 2000)
Arnolds DE,Moskowitz IP
36-4900 was used in immunohistochemistry to use transgenic mice to study the cardiac conduction system.
Tue Nov 01 00:00:00 EDT 2011
Transcriptional suppression of connexin43 by TBX18 undermines cell-cell electrical coupling in postnatal cardiomyocytes.
The Journal of biological chemistry
Kapoor N,Galang G,Marbán E,Cho HC
Published figure using Connexin 40 polyclonal antibody (Product # 36-4900) in Western Blot
Fri Apr 22 00:00:00 EDT 2011
MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.
The Journal of clinical investigation
Callis TE,Pandya K,Seok HY,Tang RH,Tatsuguchi M,Huang ZP,Chen JF,Deng Z,Gunn B,Shumate J,Willis MS,Selzman CH,Wang DZ
36-4900 was used in western blot to study the function and molecular mechanisms of the miR-208 family of miRNAs in adult mouse heart physiology
Tue Sep 01 00:00:00 EDT 2009