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Zitierungen und Referenzen (33)
Invitrogen™
Vybrant™ Cell Adhesion Assay Kit
Das Vybrant™ Zelladhäsionsassay-Kit ist ein schneller und empfindlicher Assay zur Messung der Zell-zu-Zell- oder Zelloberflächenadhäsion bei einer Vielzahl von Zelltypen.Weitere Informationen
| Katalognummer | Menge |
|---|---|
| V13181 | 1 Kit |
Katalognummer V13181
Preis (EUR)
696,00
1 kit
Menge:
1 Kit
Preis (EUR)
696,00
1 kit
Das Vybrant™ Zelladhäsionsassay-Kit ist ein schneller und empfindlicher Assay zur Messung der Zell-zu-Zell- oder Zelloberflächenadhäsion bei einer Vielzahl von Zelltypen. In diesem Assay werden Zellen mit Calcein AM markiert und dürfen dann adhärieren. Nach der Entfernung der nicht adhärenten Zellen wird die Anzahl adhärenter Zellen anhand der Calcein-Fluoreszenz berechnet.
Nur für Forschungszwecke. Nicht zur Verwendung bei diagnostischen Verfahren.
Specifications
NachweisverfahrenFluoreszent
FarbstofftypAndere Etiketten oder Farbstoffe
Format96-Well Platte
Menge1 Kit
VersandbedingungRaumtemperatur
Zur Verwendung mit (Anwendung)Zelladhäsionsassay
Zur Verwendung mit (Geräte)Mikrotiterplatten-Lesegerät
ProduktlinieVybrant
ProdukttypFärben
Unit Size1 kit
Inhalt und Lagerung
In einem Tiefkühlgerät (-5 bis -30 °C) lagern und vor Licht schützen.
Häufig gestellte Fragen (FAQ)
What are the fluorescence excitation/emission maxima for the Calcein AM and SYTOX Green nucleic acid stain provided in the Vybrant Cell Adhesion Assay Kit?
Zitierungen und Referenzen (33)
Zitierungen und Referenzen
Abstract
Effect of C-reactive protein on gene expression in vascular endothelial cells.
Journal:Am J Physiol Heart Circ Physiol
PubMed ID:15591095
'C-reactive protein (CRP) is significantly associated with the risk of ischemic cardiovascular disease in epidemiological studies. To explore if CRP has a functional role, we investigated its effect on the gene expression profile of vascular endothelial cells. Human vascular endothelial cells (human umbilical vein endothelial cells and human aortic endothelial
Atherogenic lipids induce adhesion of human coronary artery smooth muscle cells to macrophages by up-regulating chemokine CX3CL1 on smooth muscle cells in a TNFalpha-NFkappaB-dependent manner.
Journal:J Biol Chem
PubMed ID:17456471
'Recent genetic evidence has implicated the adhesive chemokine CX3CL1 and its leukocyte receptor CX3CR1 in atherosclerosis. We previously proposed a mechanism involving foam cell anchorage to vascular smooth muscle cells because: 1) CX3CL1 and CX3CR1 are expressed by both cell types in mouse and human atherosclerotic lesions; 2) foam cells
ROCK inhibitor improves survival of cryopreserved serum/feeder-free single human embryonic stem cells.
Journal:Hum Reprod
PubMed ID:19056770
'Efficient slow freezing protocols within serum-free and feeder-free culture systems are crucial for the clinical application of human embryonic stem (hES) cells. Frequently, however, hES cells must be cryopreserved as clumps when using conventional slow freezing protocols, leading to lower survival rates during freeze-thaw and limiting their recovery and growth
CXCL16 signals via Gi, phosphatidylinositol 3-kinase, Akt, I kappa B kinase, and nuclear factor-kappa B and induces cell-cell adhesion and aortic smooth muscle cell proliferation.
Journal:J Biol Chem
PubMed ID:14625285
'CXCL16, a recently discovered transmembrane chemokine, is expressed in human aortic smooth muscle cell (ASMC). It facilitates uptake of low density lipoproteins by macrophages, resulting in foam cell formation. However, it is not known whether ASMC express CXCR6, the receptor for CXCL16, or whether CXCL16 affects ASMC biology. To dissect
Targeting PKC in multiple myeloma: in vitro and in vivo effects of the novel, orally available small-molecule inhibitor enzastaurin (LY317615.HCl).
Journal:Blood
PubMed ID:17023575
'In multiple myeloma (MM) protein kinase C (PKC) signaling pathways have been implicated in cell proliferation, survival, and migration. Here we investigated the novel, orally available PKC-inhibitor enzastaurin for its anti-MM activity. Enzastaurin specifically inhibits phorbol ester-induced activation of PKC isoforms, as well as phosphorylation of downstream signaling molecules MARCKS